Sporogenesis and Toxin a Production by Clostridium Difficile

References

1. Banno Y., Kobayashi T., Kono H., Watanabe K., Ueno K., Nozawa Y. 1984; Biochemical characterization and biologic actions of two toxins (D-1 and D-2) from Clostridium difficile. Reviews of Infectious Diseases 6 (suppl. 1) 511–520
   [Google Scholar]
2. Bartlett J. G., Chang T. W., Gurwith M., Gorbach S. L., Onderdonk A. B. 1978; Antibiotic-associated pseudomembranous colitis due to toxin-producing clostridia. New England Journal of Medicine 298:531–534
   [Google Scholar]
3. Borriello S. P., Honour P. 1981; Simplified procedure for the routine isolation of Clostridium difficile from faeces. Journal of Clinical Pathology 34:1124–1127
   [Google Scholar]
4. Burdon D. W., Thompson H., Candy D. C. A., Kearns M., Lees D., Stephen J. 1981; Enterotoxin(s) of Clostridium difficile. Lancet 2:258–259
   [Google Scholar]
5. Duncan C. L., Strong D. H. 1968; Improved medium for spoliation of Clostridium perfringens. Applied Microbiology 16:82–89
   [Google Scholar]
6. Florin I., Thelestam M. 1981; Intoxication of cultured human lung fibroblasts with Clostridium difficile toxin. Infection and Immunity 33:67–74
   [Google Scholar]
7. Frieben W. R., Duncan C. L. 1973; Homology between enterotoxin protein and spore structural protein in Clostridium perfringens Type A. European Journal of Biochemistry 39:393–401
   [Google Scholar]
8. George R. H., Symonds J. M., Dimock P., Brown J. D., Arabi Y., Shinagawa N., Keighley M. R. B., Alexander-Williams J., Burdon D. W. 1978; Identification of Clostridium difficile as a cause of pseudomembranous colitis. British Medical Journal 1:695
   [Google Scholar]
9. Gould G. W. 1971; Methods for studying bacterial spores. In Norris J. R., Ribbons D. W. (eds) Methods in microbiology 6A. chapter VI Academic Press; London: pp 327–381
   [Google Scholar]
10. Justus P. G., Martin J. L., Goldberg D. A., Taylor N. S., Bartlett J. G., Alexander R. W., Mathias J. R. 1982; Myoelectric effects of Clostridium difficile’. Motility-altering factors distinct from its cytotoxin and enterotoxin in rabbits. Gastroenterology 83:836–843
    [Google Scholar]
11. Ketley J. M., Haslam S. C., Mitchell T. J., Stephen J., Candy D. C. A., Burdon D. W. 1984; Production and release of toxins A and B by Clostridium difficile. Journal of Medical Microbiology 18:385–391
    [Google Scholar]
12. Lyerly D. M., Lockwood D. E., Richardson S. H., Wilkins T. D. 1982; Biological activities of toxins A and B of Clostridium difficile. Infection and Immunity 35:1147–1150
    [Google Scholar]
13. Redmond S. C., Ketley J. M., Mitchell T. J., Stephen J., Burdon D. W., Candy D. C. A. 1985; Detection of Clostridium difficile enterotoxin (toxin A) by ELISA and other techniques. In Collins C. H., Grange J. M. (eds) Isolation and identification of micro-organisms of medical and verterinary importance. Society of Applied Bacteriology, Technical Series 21 Academic Press; London:237–250
    [Google Scholar]
14. Taylor N. S., Thorne G. M., Bartlett J. G. 1980; Separation of an enterotoxin from the cytotoxin of Clostridium difficile. Clinical Research 28:285A
    [Google Scholar]
15. Taylor S. N., Thorne M. G., Bartlett J. G. 1981; Comparison of two toxins produced by Clostridium difficile. Infection and Immunity 34:1036–1043
    [Google Scholar]
16. Wilson K. H., Kennedy M. J., Fekety F. R. 1982; Use of sodium taurocholate to enhance spore recovery on a medium selective for Clostridium difficile. Journal of Clinical Microbiology 15:443–446
    [Google Scholar]
17. Yamakawa K., Nishida S., Nakamura S. 1983; C; toxicity in extract of Clostridium hotulinum Type C spores. Infection and Immunity 41:858–860
    [Google Scholar]